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Review Question - QID 104276

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QID 104276 (Type "104276" in App Search)
A 54-year-old man presents with 3 days of non-bloody and non-bilious emesis every time he eats or drinks. He has become progressively weaker and the emesis has not improved. He denies diarrhea, fever, or chills and thinks his symptoms may be related to a recent event that involved sampling many different foods. His temperature is 97.5°F (36.4°C), blood pressure is 133/82 mmHg, pulse is 105/min, respirations are 15/min, and oxygen saturation is 98% on room air. Physical exam is notable for a weak appearing man with dry mucous membranes. His abdomen is nontender. Which of the following laboratory changes would most likely be seen in this patient?

Anion gap metabolic acidosis and hypokalemia

2%

1/54

Metabolic alkalosis and hyperkalemia

9%

5/54

Metabolic alkalosis and hypokalemia

20%

11/54

Non-anion gap metabolic acidosis and hypokalemia

9%

5/54

Respiratory acidosis and hyperkalemia

57%

31/54

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This patient is experiencing severe vomiting likely secondary to food poisoning. He would likely have hypokalemia and a metabolic alkalosis secondary to activation of the renin-angiotensin-aldosterone system (RAA) and HCl loss in his vomit.

Vomiting can cause dehydration and electrolyte abnormalities. When patients vomit, they lose both chloride and acid equivalents. For this reason, vomiting over long periods of time can lead to metabolic alkalosis and hypochloremia. Additionally, patients with vomiting are typically dehydrated which stimulates the renin-angiotensin-aldosterone system which subsequently causes retention of sodium, chloride, and water. Activation of the RAA system also leads to the loss of potassium secondary to aldosterone's activity on the principal cells and loss of hydrogen ions secondary to its action on the intercalated cells. No specific treatment is needed for these patients other than fluids and electrolyte replacement.

Incorrect Answers
Answer 1: Anion gap metabolic acidosis and hypokalemia would be seen in diabetic ketoacidosis. Patients will experience an anion gap acidosis from fatty acid breakdown (from insulin deficiency), hyperventilation (lowering the PCO2 to correct the pH), and hypokalemia late in presentation as total body potassium stores continue to deplete.

Answer 2: Metabolic alkalosis and hyperkalemia are incorrect as aldosterone causes potassium secretion from the principal cells leading to hypokalemia.

Answer 4: Non-anion gap metabolic acidosis and hypokalemia are incorrect as increased aldosterone activity leads to H+ wasting from the intercalated cells causing metabolic alkalosis.

Answer 5: Respiratory acidosis and hyperkalemia are incorrect as this patient's HCl loss in his vomit will induce a metabolic alkalosis which will lead to respiratory compensation to correct the acid-base disturbance (in the form of an elevated PCO2); however, aldosterone's action on the principal cells leads to potassium loss in the urine causing hypokalemia.

Bullet Summary:
Vomiting leads to HCl loss and activation of the renin-angiotensin-aldosterone system which can cause metabolic alkalosis and hypokalemia.

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